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Your skin cares for you, too. Your skin is designed to protect you from external harm

If you’re like us, then you probably find joy in caring for your skin. You might also know about all the different treatments available that can help you feel glowy, clean and satisfied with your skin’s appearance. But, do you ever stop and reflect on how well designed your skin is to protect you? 

Let’s break down the basic anatomy of the skin and how it works to protect you from external factors like temperature changes, pollution, and harsh chemicals. The skin is comprised of three layers. And these layers are made up of cells and functions that are actively working to ensure the health and safety of your skin. 

Layer 1: Epidermis  

This is your body’s protective wrap and the layer that is in permanent contact with the outside world. It’s also waterproof and acts as a barrier against microbes (disease-causing bacteria).  The epidermis is made up of two main cell types: Keratinocytes and Melanocytes 

Keratinocytes make up a majority of the cells in the epidermis and form the barrier that protects against environmental damages like bacteria and viruses, heat, UV radiation, and water loss.

Melanocytes are your melanin-producing cells. Melanin is the pigment that gives our eyes, hair, and skin it’s characteristic colour.

Layer 2: Dermis   

This flexible but tough support structure cushions your body from stress and strain. The major players in the dermis are cells called fibroblasts which synthesize the proteins to create the extracellular matrix.  

Is this getting too scientific for you? Bear with us, we promise you’ll like this next part – the main proteins in the extracellular matrix are collagen and elastin . Ever heard of them? So many products promise to boost collagen and restore elasticity resulting in a firmer, younger looking appearance. But what role do these proteins play in your skin? 

Collagen provides structural support to cells, meaning the more collagen present, the more supple and healthy skin appears.  

Elastin gives skin it’s elasticity, allowing it to stretch and subsequently return to its normal state.  

Both proteins play critical roles in wound healing and skin ageing and are found in many skin treatments available today with varying degrees of effectiveness.  

Layer 3: Subcutis 

Fibroblasts are also found in the lowermost layer of your skin, along with a fat layer that separates your dermis from the muscles. These fat cells also insulate your body, and cushion and protect your skin. 

If any of these areas of your skin experience dysfunction or harm, it can lead to conditions like rosacea and acne. And of course, skin also undergoes the ageing process as you grow older, which is as natural as it is inevitable.  

We also believe in working with your skin to help combat any issues or dysfunctions that may arise, because we know that chronic skin issues happen for a variety of reasons and you are certainly not to blame. Kleresca® treatments stimulate your skin’s own repair mechanisms, helping you achieve a balanced and healthy skin, all year round. 

You, your skin, and Kleresca®. Skincare dream team? We think so.

 

Follow us on our social media channels to stay up to date with our latest news, real-life results stories, and breakthrough scientific advancements in treating the skin. 

      
        




Sources

Weller, R. et al. 2015 Clinical Dermatology. Fifth Edition. Wiley Blackwell 
Buzney, E. et al. Basic Science of the Skin Module 2008 – 2012. American Academy of Dermatology. 
Elias, P.M. 2007 The skin barrier as an innate immune element. Seminars in Immunopathology. 29(1): 3–14.  McGrath JA; et al. 2004. Anatomy and Organization of Human Skin. In Burns T et al. Rook’s Textbook of Dermatology, 7th Ed.  
Ferreira dos Santos, I. et al. Mechanisms regulating melanogenesis. An Bras Dermatol. 2013 Jan-Feb; 88(1): 76–83. 
D’Mello, S. et al. 2016. Signaling Pathways in Melanogenesis. Int. J. Mol. Sci.  17, 1144. 
Marks et al. 2006. Lookingbill and Marks' Principles of Dermatology (4th ed.). Elsevier Inc. Page 8–9. 
Chung JH et al. 2001. Modulation of Skin Collagen Metabolism in Aged and Photoaged Human Skin In Vivo. J Invest Dermatol. Nov;117(5):1218-24 
Moronkeji K. & Akhtar R. 2015. Mechanical Properties of Aging Human Skin. In: Derby B., Akhtar R. (eds) Mechanical Properties of Aging Soft Tissues. Engineering Materials and Processes. Springer, Cham 
Nielsen, M.E., et al (2017). Introducing: photobiomodulation by low energy chromophore-induced fluorescent energy. Mechanisms of Photobiomodulation Therapy IV, SPIE Photonics West BIOS, San Francisco, 2017; 28 January – 2 February 
Nielsen, Schoedt & Bak-Christensen. Clinical Evaluation – Kleresca® Biophotonic Treatments. 2017. Kleresca® Data on File   

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